1434 680 1025 11 16087 document 1025 JCF2007_(3).pdf application/pdf 244293 2011-09-09 10:21:39 https://vuir.vu.edu.au/1434/1/JCF2007_%283%29.pdf 1434 1 application/pdf text en public

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JCF2007_(3).pdf

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http://eprints.org/relation/isVersionOf https://vuir.vu.edu.au/id/document/1025 http://eprints.org/relation/isVolatileVersionOf https://vuir.vu.edu.au/id/document/1025 http://eprints.org/relation/ismediumThumbnailVersionOf https://vuir.vu.edu.au/id/document/1025 112955 2 2557808 document 112955 small.jpg image/png 4ea6254526bf2838b7b4e8361d81c037 MD5 1618 2023-12-17 13:21:21 https://vuir.vu.edu.au/1434/7/small.jpg 1434 7 7 image/png other Thumbnails conversion from text to thumbnail_small en public

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http://eprints.org/relation/isVersionOf https://vuir.vu.edu.au/id/document/1025 http://eprints.org/relation/isVolatileVersionOf https://vuir.vu.edu.au/id/document/1025 http://eprints.org/relation/issmallThumbnailVersionOf https://vuir.vu.edu.au/id/document/1025 archive 292 disk0/00/00/14/34 2008-12-04 01:54:34 2021-08-27 13:58:26 2008-12-04 01:54:34 article show Williams Andrew D Carey Michael F Selig Steve E Selig, Steve E Hayes Alan 0000-0003-1398-3694 Krum Henry Patterson Jeremy Toia Deidre Hare David L pub 320000 BiomedicalSciences exercise, oxidative capacity, skeletal muscle Background. We aimed to determine the role of skeletal muscle mitochondrial ATP production rate (MAPR) in relation to exercise tolerance following resistance training in CHF. Methods and Results. Thirteen CHF patients (NYHA functional class 2.3 ± 0.5; LVEF 26 ± 8%; age 70 ± 8 years) underwent testing for VO2peak, and resting vastus lateralis muscle biopsy. Patients were then randomly allocated to 11 weeks of RT, (n = 7) or continuance of usual care (C, n = 6) following which testing was repeated. Muscle samples were analysed for MAPR, metabolic enzyme activity and capillary density. VO2peak and MAPR in the presence of the pyruvate and malate (P+M) substrate combination, representing carbohydrate metabolism, increased in RT (p<0.05) and decreased in C (p<0.05) with a significant difference between groups (VO2peak p = 0.005; MAPR p = 0.03). There was a strong correlation between the change in MAPR and the change in VO2peak over the study (r = 0.875; p < 0.0001), the change in MAPR accounting for 70% of the change in VO2peak. Conclusions. These findings suggest that mitochondrial ATP production is a major determinant of aerobic capacity in CHF patients and can be favourably altered by muscle trengthening exercise. 2007 published Elsevier public Journal of Cardiac Failure 13 2 79-85 TRUE 1071-9164 Includes bibliographical references: pp. 19-22 FALSE 57 2-s2.0-33947518543 2023-07-02 04:00:17