α-actinin-3 deficiency is associated with increased exercise performance and training-induced changes in mitochondrial respiration in humans

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Yan, Xu ORCID logoORCID: https://orcid.org/0000-0001-8547-4210, Eynon, Nir ORCID logoORCID: https://orcid.org/0000-0003-4046-8276, Papadimitriou, Ioannis D, Kuang, Jujiao ORCID logoORCID: https://orcid.org/0000-0002-1366-0089, Tirosh, Oren, O'Keefe, Lannie ORCID logoORCID: https://orcid.org/0000-0002-0264-0007, Anderson, Mitchell, North, Kathryn N and Bishop, David ORCID logoORCID: https://orcid.org/0000-0002-6956-9188 (2015) α-actinin-3 deficiency is associated with increased exercise performance and training-induced changes in mitochondrial respiration in humans. In: Experimental Biology 2015, 28 March 2015-01 April 2015, Boston.

Abstract

Publication is Abstract only. Deficiency of α-actinin-3 protein, as a consequence of the ACTN3 577XX genotype, has been shown to influence exercise performance. However, little is known about how α-actinin-3 deficiency influences training response in humans.

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Additional Information

Paper 677.27
Item type Conference or Workshop Item (Paper)
URI https://vuir.vu.edu.au/id/eprint/30427
DOI 10.1096/fasebj.29.1_supplement.677.27
Subjects Historical > FOR Classification > 1106 Human Movement and Sports Science
Historical > FOR Classification > 1116 Medical Physiology
Current > FOR (2020) Classification > 4207 Sports science and exercise
Historical > Faculty/School/Research Centre/Department > Institute of Sport, Exercise and Active Living (ISEAL)
Keywords High-Intensity Interval Training; alpha-actinin-3; graded exercise test; peak oxygen uptake; Lactate threshold, proteins, exercise performance, respiration, training induced changes
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